Abstract
Acute transmural ischaemia often shortens ventricular repolarization and increases repolarization dispersion, leading to life threatening ventricular arrhythmias in animal models and human subjects. Experimental studies and clinical observations have shown that acute subendocardial ischaemia rarely causes serious ventricular arrhythmia. We hypothesized that the different arrhythmia outcomes between transmural and subendocardial ischaemia are largely due to the homogenous prolongation in ventricular repolarization after acute subendocardial ischaemia.
Original language | English |
---|---|
Pages (from-to) | 137-139 |
Number of pages | 3 |
Journal | Medical Hypotheses |
Volume | 68 |
Issue number | 1 |
DOIs | |
Publication status | Published - 2006 |