and immediately post-recovery, 2-h, 24-h and 48-h post-recovery. The results indicated that despite a post-exercise suppression in voluntary torque, a recovery of COLD did not significantly enhance maximal voluntary torque (MVC) and activation (VA) (P>0.05). Further, post-exercise elevations in creatine kinase (CK), aspartate aminotransferase (AST) and c-reactive protein (CRP) were not significantly altered by COLD (P>0.05); although, perceptions of pain were lower 48-h post-recovery following COLD compared to CONT. Therefore, when explicit eccentric muscle actions, isolated to a single-leg, elicited prolonged muscle damage, post-exercise COLD did not hasten the recovery of skeletal muscle function despite enhancing perceptions of pain.Study two examined the effects of CWI following simulated team-sport exercise in the heat. Two sessions of a 2 x 30-min intermittent-sprint protocol (ISE) in 32ËšC and 52% humidity were performed, followed by a 20-min CWI or passive recovery (CONT). Neuromuscular function, perceived MS, CK, AST and CRP were measured pre- and post-exercise, and immediately post-recovery, 2-h and 24-h post-recovery; whilst core temperature (Tcore), heart rate (HR), perceptions of exertion, thermal strain and thirst were recorded pre- during and post-exercise. MVC and VA were reduced post-exercise in both conditions and remained suppressed for the 24-h recovery period (P<0.05), whilst CK, AST and CRP were elevated above pre-exercise values (P<0.05). Implementation of post-exercise CWI produced a more rapid rate of reduction in Tcore, HR and MS, whilst increasing post-recovery MVC, VA and root mean square (RMS) of the electromyogram (EMG) signal (P<0.05). In contrast, MVC and RMS measured at 24-h post-recovery were significantly higher in CONT compared to CWI (P=0.05).
|Qualification||Doctor of Philosophy|
|Award date||01 Aug 2011|
|Place of Publication||Australia|
|Publication status||Published - 2011|